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2.3 Others In addition to A deposition and tau protein hyper- phosphorylation, pathological changes such as neuroinflammation and mitochondrial dysfunction are closely related to AD pathogenesis

Central behavioral effects were subsequently shown to be abolished by a non-selective MC3R/MC4R antagonist, confirming receptor-mediated central MCR activation (PMC PMC1572913)

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Residues are show in paratheses