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Part of the problem is we dont have research on long-term impacts yet, nor is the drug available at such a scale that those impacts could be seen yet

A related possibility is that some hindbrain neurons express both the estrogen receptor and GLP1R, and that activation of the former (by estrogen) amplifies signal transduction of the latter (by a GLP1R agonist)

use with caution in those with cardiovascular conditions [1]