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It is investigational (not FDA-approved on its own)

NF-B, the master transcriptional regulator of pro-inflammatory gene expression, is activated downstream of TNF receptor and IL-1 receptor signaling

corpus callosum in CAE), in the former, a deliberately orchestrated primary autoimmune process directed at myelin antigens results in lymphocytic infiltration, secondary injury to white matter and a universal innate response, whereas in CAE, where the primary insult is a biochemical one deliberately targeting intrinsic CNS myelin, following immune stimulation, the resulting lesion is pathologically similar (Day, 2005

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