First, here's the infographic: Keep reading to see what it gets wrong
3 Mechanisms of sulforaphane-based neuroprotection 3.1 Sulforaphane and oxidative stress regulation Reactive oxygen species (ROS), such as superoxide anions and hydrogen peroxide, are byproducts of cellular metabolism and function within organelles such as mitochondria, endoplasmic reticulum, and peroxisomes ( As described above, sulforaphane modifies cysteine residues within the KEAP1 ubiquitin ligase, albeit distinct cysteine residues from those that become oxidized by ROS (Figure 2)
Its multi-target mechanisms also indicate therapeutic potential for metabolic, renal, and neurodegenerative disorders
Human clinical trial data are absent
Animal studies suggest that it reduces NNMT activity, leading to better NAD+ retention, improved metabolic efficiency, and reduced fat mass