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These findings reveal that GGT1 safeguards against CaOx-induced nephrotoxicity by targeting GCLC to regulate ferroptosis and highlight GGT1 as a potential therapeutic target in nephrolithiasis

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Eur Respir J

53 In another more recent study, IV glutathione prevented the increase in oxidative stress markers while oral NAC (N-acetyl cysteine), an amino acid needed to make glutathione, had no effect

Our findings indicated that targeting ACSL4 or, alternatively, upregulating GPX4 or FSP1 may help mitigate AKI by reducing ferroptosis, offering potential therapeutic strategies for oxalate-induced AKI