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Mineralocorticoids, whether in excess (Hyperaldosteronism) or in deficiency (Hypoaldosteronism), are linked to oxidative stress, mainly via activation of the renin-angiotensin-aldosterone system (RAAS) and disruption of cellular homeostasis due to sodium and potassium imbalances

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Circulating levels peak in early adulthood and decline significantly with age dropping from roughly 200 ng/mL at age 20 to approximately 80 ng/mL by age 60