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This microgliaastrocyte crosstalk is implicated in neurodegenerative diseases such as AD, Parkinsons disease (PD), and Huntingtons disease (HD) [93]

From these results we hypothesize that Dihexa-mediated protection relies, at least in part, on the activation of multiple downstream targets of the HGF/c-Met system (Akt-TOR and Ras-ERK)