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In research models it produces pulsatile GH release through GHS-R1a receptor activation, with a short half-life that makes it suitable for protocols requiring controlled, time-limited GH pulses

doi: 10.1111/mmi.14870 32 HillionM.AntelmannH

(4) leave the cell trough the exporter FPN ( 2+ is a potent source of oxidative damage, because Fe 2+ reacts with hydrogen peroxide (H 2 O 2 ), which generates Fe 3+ and hydroxyl radicals (OH

Y., & Park, J