Although UPR is a compensatory cellular response that reduces protein synthesis and enhances protein folding and degradation, UPR also contributes to lung cell apoptosis and lung inflammation during excessive ER stress [43,44,45,46,47]
"The biggest shock was that I was the old smoker and came out with the best lung capacity of them all."
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Smoking cessation rapidly increases circulating progenitor cells in peripheral blood in chronic smokers
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