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In contrast, TIGAR overexpression or treatment with Fer-1, a ferroptosis inhibitor, significantly increases NADPH, GPX4, and GSH levels, reduces GSSG expression, and attenuates neuronal ferroptosis and neurological impairment

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Metabolic analysis revealed no changes in the levels of metabolites in either strains, except for EGT, and hercynylcysteine sulphoxide in egt2