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Although the above mechanisms have been discovered and confirmed repeatedly, therapeutic strategies should be more specifically targeted to mitochondrial energy defects rather than the neuroprotective effects of antioxidants [52]
Until now, this hypothesis has only been substantiated by ex vivo experiments where vaginal tissue strips from POP cases were exposed to riboflavin and subsequent UVA photoactivation cells 128,129
Food and Drug Administration, is now widely used as a dietary supplement (15, 16)
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Muscle and bone loss is a growing concern
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