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By promoting immune cell migration to sites of genuine infection, it redirects immune activity away from self-tissue and toward actual threats

Third, we characterized the primary receptor for CXCL10 in the ovaries, IL18R1, and showed that it dominates over CXCR3 in binding to CXCL10

Decreased antioxidant buffering capacity allows vaccine-induced inflammation to persist at a higher level and for a longer duration

See the Symptoms & Warning Signs page for what these crises look like