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These include regulation of mechanisms controlling reactive oxygen species (ROS), which cause macromolecular damage 16,17 , mitochondrial turnover or mitophagy 18 , and the mitochondrial unfolded protein response (UPR mt ), in which mitochondrial damage can be prevented by the removal of aggregated or unfolded proteins 19

tb within in vitro granulomas when compared to healthy individuals, thus confirming previous studies demonstrating T2DM individuals have a reduced ability to control the mycobacterial infections compared to healthy individuals ( Figures 2A, B )

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