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During the activation phase, stimuli such as Nigericin (113) prompted NLRP3 to oligomerize through the NACHT structural domain, recruit apoptosis-associated speck-like protein containing a CARD (ASC) and NIMA-associated kinase 7 (NEK7), and promote ASC oligomerization to form macromolecular foci of specks, which in turn led to pro-caspase-1 recruitment and activation, and assembly to form NLRP3 inflammatory vesicles (114)

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Primary effects: Increases blood flow to injured areas More blood = more oxygen, nutrients, and growth factors reaching damaged tissue

In addition, the redox state influences the quantity of nitric oxide (NO) available for bioavailability in the body

swiftly into the body for fast change